Month: February 2014

CLEAN YOUR KIDNEYS – Natural Home Remedy

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Years pass by and our kidneys are filtering the blood by removing salt, poison and any unwanted entering our body.

With time, the salt accumulates and this needs to undergo cleaning treatments and how are we going to overcome this?

It is very easy, first take a bunch of parsley or Cilantro ( Coriander Leaves ) and wash it clean
Then cut it in small pieces and put it in a pot and pour clean water and boil it for ten minutes and let it cool

down and then filter it and pour in a clean bottle and keep it inside refrigerator to cool.

Drink one glass daily and you will notice all salt and other accumulated poison coming out of your kidney by

urination also you will be able to notice the difference which you never felt before.

parsley or Cilantro ( Coriander Leaves ) is known as best cleaning treatment for kidneys and it is natural!

Source : facebook


Texila American University Offers Medicine & Nursing Programs At Affordable Cost

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tauedu is an educational website that provides a complete list of medical schools in the Caribbean. This website also provides details about the clinical transfers, exams and other related queries in brief

Here is a fact that many students choose medical schools in the Caribbean because the cost of living and the tuition fee of the program is comparatively low when compared to US and Canadian medical schools.

Texila American University is one of the best medical schools in the Caribbean located in Guyana, provides medicine and nursing programs at affordable cost. The main aim of the university is to provide quality of education at an affordable cost. Students will get an opportunity to do their clinical rotations in United States

TAU offers Doctor of Medicine programs for 4 and 5.5 years based on the eligibility of the students. Students may opt to choose normal track or US track. The eligibility of 4 year doctor of medicine program is completion of diploma or bachelors degree and for 5.5 year program the eligibility is completion of high school

TAU College of Nursing provides 4 years Bachelor of Science in Nursing. Fee of the program is comparatively low compared to other Caribbean medical schools. The university is partnered with the University of West England UK, to provide a world class education in nursing

About is an educational portal that brings all university details together that is located in the Caribbean region. Caribbean Medical Schools teach, train or conduct clinical research for various health care programs which includes nursing, medicine, dentistry and pharmacy.

The Caribbean medical is unique in bringing together the Medical Sciences sector, to discuss emerging issues in the delivery of world-class healthcare education related to Medical research and service.

The universities in the Caribbean also accept the transfer students instead of passing rates. so this might be a option for many students who still try to continue with a healthcare profession. Here is a fact to prove you. The New York Times reports that 25 percent of the citizens are trained and qualified from overseas. Many of these foreign-trained citizens went to a Caribbean Medical Schools.

As an organization it uses up a unique position, adopting undergraduate and post graduate medical courses, health-related research, a critical interface with the wellness service, and several postgraduate knowledge training

Sitting linked to increased risk for disability, study shows

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For people over age 60, every extra hour of the day spent sitting is linked to an increased risk for developing life-altering physical disabilities, according to a new study from Northwestern University’s Feinberg School of Medicine.
Researchers have long known that being sedentary is harmful to a person’s health – and that moderate activity has numerous health benefits.

“But what we did not know is whether those were just two ways of looking at the same question – that is, if being sedentary meant you had insufficient activity, or whether it was a separate risk factor,” study author Dorothy Dunlop, a professor of medicine and preventive medicine .“And this study is a smoking gun that being sedentary is a separate risk factor.”

In a study published in the Journal of Physical Activity and Health, Dunlop analyzed data collected from 2,286 adults over age 60 who had participated in the National Health and Nutrition Examination Survey. From 2002 to 2005, each study participant wore an accelerometer – a device that monitors physical activity – to objectively assess their levels of activity, or inactivity.
Researchers also gathered health data from the participants to determine their risk for disability.

“The way they defined disability was limitations in basic activities you need to be able to do to stay independent – feeding yourself, bathing yourself, dressing yourself, walking from room to room,” Dunlop said.

Overall, the researchers found that for every extra hour per day that a person spent sitting, their risk for disability increased by 50 percent.

“If you take two 65-year-old women, with the same health profiles, and…one is sitting or doing very little about 12 hours a day, her chance of being in the disabled pool is about 6 percent,” Dunlop said. “If you take another person, also 65 years old, same health profile, but she sits for 13 hours a day, her chance of being disabled is 9 percent; it’s an increase of 50 percent for each hour.”
Furthermore, among people who spent the most time sitting, their increased risk for disability could not be explained by a lack of exercise. However, Dunlop cautioned that this doesn’t mean people should give up and quit the gym.

“There are two messages here – being physically active is very important, it does help you and it’s well documented that it reduces your risk of disability,” Dunlop said. “Being sedentary is a separate risk factor. You want to focus on both – be as active as possible and for people…who have desk jobs and sit [for] a large portion of daylight hours, it is beneficial to find opportunities to replace some of that sitting with other activities.”

Though the study did not examine the mechanisms behind the link between sitting and the increased risk for disability, Dunlop did have some theories.

“My clinical colleagues tell me that when a person sits for an extended time, that their muscles burn less fat and the blood is flowing more sluggishly,” Dunlop said. “And furthermore, if someone slumps in their chair then their back and stomach muscles go unused and the issue of idle muscles and slow circulation can contribute to all kinds of chronic diseases like high blood pressure, heart disease, diabetes, and also contribute to varicose veins, swollen legs and ankles.”

And though this study focused on an older population, Dunlop said the health effects of sitting on a younger population are likely similar.

“We did not look at people under age 60 because disability is so infrequent in that younger age group,” Dunlop said. “However, there is no reason to expect their experience would be different.”

Further research will be needed to definitively prove the link between sitting and an increased risk for disability – but until then, Dunlop advised people to try to replace sedentary time with light physical activity as often as possible.

“When I talk on the telephone, if I just stand up while I’m talking, that breaks up my sitting. When I go to the grocery store, if I take a walk around the store first before I start shopping, [that helps],” Dunlop said. “What you’re trying to do is simply accumulate more time where you’re not sitting or being sedentary or parked in front of a computer or television.”


Your diet may not fit your genes, scientists say

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You are what you eat, and what you eat could be making you age prematurely; in fact, it may even be killing you.

And it’s not all about 64-ounce cups of sugary soda pop. It may just be that those skinny jeans don’t fit your genes.

Your diet can trigger genetic effects that cause you to age more rapidly, according to a recently completed study at the University of Southern California Davis. The study’s authors, Sean Curran and Shanshan Pang, identified a collection of genes that allow an organism to adapt to different diets and showed that without them, even minor dietary changes can cause premature aging, even death.

The researchers also revealed a reason why some diets work for specific groups of people and are ineffective for others.

In the study, for example, the scientists identified a gene called alh-6 that delayed the effects of aging depending on what type of diet a worm was fed, by protecting it against diet-induced defects. The worm, known as the Caenorhabditis elegans, has been used in genetic research for decades because many of its genes have similar functions in humans. But the researchers do not know precisely what element of the worm’s diet triggered the mitochondrial defects that resulted in advanced aging.

So don’t expect to see personalized prescription diets soon, though related research indicates this may eventually happen.

Earlier studies have contributed to the area of nutrigenetics, which examines the role of genetic variation on interactions between diet and health. A study last year looked at 3,500 people and the two expressions of a gene, the C and T versions of APOA2, and found that people who had the C version of the gene and ate a diet high in saturated fats gained weight. But those with the T version were less vulnerable to the high-fat diet.

Genetics are not a get-out-of-fat-free card, though. When both groups ate a healthier low-fat diet, they both benefited with a lower body mass index reading.

Nevertheless, the day could arrive when doctors tell some patients that it’s safe to eat doughnuts and fried foods, while others have to stick to Brussels sprouts and kale.

Curran points out that full genome sequencing is available now for less than $1,000, meaning it’s possible to start doing clinical studies of specific diets and genetic markers.

“Imagine one day,” he said, “when an infant is born, its genome is sequenced and optimal diets can be predicted accurately.”


Glaucoma Linked to New Eye Layer

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The findings, published in a paper in the BritishJournal of Ophthalmology, could shed new light on glaucoma, a devastating disease caused bydefective drainage of fluid from the eye and the world’s second leading cause of blindness.

The latest research shows that the new layer, dubbed Dua’s Layer after the academic ProfessorHarminder Dua who discovered it, makes an important contribution to the sieve-like meshwork, thetrabecular meshwork (TM), in the periphery of the cornea.

The TM is a wedge-shaped bandof tissue that extends along the circumference of the angle of the anterior chamber of the eye.It is made of beams of collagen wrapped in a basement membrane to which trabecular cells andendothelial cells attach. The beams branch out randomly to form a ‘meshwork’.

Pressurewithin the eye is maintained by the balance of aqueous fluid production by eye tissue called theciliary body and drainage principally through the TM to the canal of Schlemm, a circular channelin the angle of the eye.

Defective drainage through the TM is an important cause ofglaucoma, a condition that leads to raised pressure in the eye that can permanently affectsight. Around 1 to 2% of the world’s population yearly have chronic glaucoma and globally around45 million people have open angle glaucoma which can permanently damage the optic nerve — 10% ofwhom are blind.

The latest research by Professor Dua and colleagues in AcademicOphthalmology at The University of Nottingham sheds new light on the basic anatomy of Dua’sLayer, which is just 15 microns thick but incredibly tough. Comprised of thin plates ofcollagen, it sits at the back of the cornea between the corneal stroma and Descemet’smembrane.

By examining human donor eyes using electron microscopy, the researchers wereable to look at Dua’s Layer beyond the central part of the cornea to shed more light on itsfeatures at the extreme periphery of the cornea. They discovered that the collagen fibres ofDua’s Layer also branch out to form a meshwork and that the core of TM is in fact an extensionof Dua’s Layer.

It is hoped the discovery will offer new clues on why the drainage systemmalfunctions in the eyes of some people, leading to high pressure.

Professor Dua said:”Many surgeons who perform lamellar corneal transplant recognise this layer as an important

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Foetus With 2 Brains, 2 Faces: Docs Say Abort, Parents Say No

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Doctors have advised them to abort their foetus which has 2 faces and 2 brains, but this Australian couple says they plan to go ahead with the pregnancy and never even thought about abortion.

The unborn child’s father Simon Howie said, “It’d be the same as being a child with autism or Down syndrome. I don’t believe in terminating the baby if it’s healthy and growing fine, and everything is going to plan.”

Three-dimensional sonograms show the child with two legs, two arms, one body and all vital organs, including a strong heart. But the child has two faces with the same features and two brains joined by a brain stem. When the doctors discovered the condition and asked the parents to abort the child, it was too late for a traditional abortion.

“It’s probably the rarest of all the conjoined twins. You’d be thinking numbers of one in a million to one in two million for this kind of anomaly,” Dr Greg Kesby, a maternal foetal specialist, told MSN.

The couple already has seven kids and are ready to take on all the challenges and medical treatment for the happiness of the eighth child. Dr Greg Kesby said that there were chances that the baby might not be able to survive till the full term and if it survives the treatment will be very expensive.

This condition of rare duplication is known as craniofacial hyperhidrosis or diprosopus and till date only 35 cases have ever been recorded. And out of these, no one was able to survive.

Mother of the foetus Renee Young suffers from rheumatoid arthritis and was 15 weeks and two days pregnant when the doctors informed her about the rare condition.

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White Matter ‘scaffold’ of Human Brain Identified

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Neuroscientists have now identified he critical communications network that supports brain function- the white matter “scaffold” of the human brain.

Their work, published Feb. 11 in the open-source journal Frontiers in Human Neuroscience, has major implications for understanding brain injury and disease. By detailing the connections that have the greatest influence over all other connections, the researchers offer not only a landmark first map of core white matter pathways, but also show which connections may be most vulnerable to damage.

“We coined the term white matter ‘scaffold’ because this network defines the information architecture which supports brain function,” said senior author John Darrell Van Horn of the USC Institute for Neuroimaging and Informatics and the Laboratory of Neuro Imaging at USC.

“While all connections in the brain have their importance, there are particular links which are the major players,” Van Horn said.

Using MRI data from a large sample of 110 individuals, lead author Andrei Irimia, also of the USC Institute for Neuroimaging and Informatics, and Van Horn systematically simulated the effects of damaging each white matter pathway.

They found that the most important areas of white and gray matter don’t always overlap. Gray matter is the outermost portion of the brain containing the neurons where information is processed and stored. Past research has identified the areas of gray matter that are disproportionately affected by injury.

But the current study shows that the most vulnerable white matter pathways – the core “scaffolding” – are not necessarily just the connections among the most vulnerable areas of gray matter, helping explain why seemingly small brain injuries may have such devastating effects.

“Sometimes people experience a head injury which seems severe but from which they are able to recover. On the other hand, some people have a seemingly small injury which has very serious clinical effects,” says Van Horn, associate professor of neurology at the Keck School of Medicine of USC. “This research helps us to better address clinical challenges such as traumatic brain injury and to determine what makes certain white matter pathways particularly vulnerable and important.”

The researchers compare their brain imaging analysis to models used for understanding social networks. To get a sense of how the brain works, Irimia and Van Horn did not focus only on the most prominent gray matter nodes – which are akin to the individuals within a social network. Nor did they merely look at how connected those nodes are.

Rather, they also examined the strength of these white matter connections, i.e. which connections seemed to be particularly sensitive or to cause the greatest repercussions across the network when removed. Those connections which created the greatest changes form the network “scaffold.”

“Just as when you remove the internet connection to your computer you won’t get your email anymore, there are white matter pathways which result in large scale communication failures in the brain when damaged,” Van Horn said.

When white matter pathways are damaged, brain areas served by those connections may wither or have their functions taken over by other brain regions, the researchers explain. Irimia and Van Horn’s research on core white matter connections is part of a worldwide scientific effort to map the 100 billion neurons and 1,000 trillion connections in the living human brain, led by the Human Connectome Project and the Laboratory of Neuro Imaging at USC.

Irimia notes that, “these new findings on the brain’s network scaffold help inform clinicians about the neurological impacts of brain diseases such as multiple sclerosis, Alzheimer’s disease, as well as major brain injury. Sports organizations, the military and the US government have considerable interest in understanding brain disorders, and our work contributes to that of other scientists in this exciting era for brain research.”